NA-Semax-Amidate

Semax, an analogue of adrenocorticotropin (4-10), is a potential agent for the treatment of attention-deficit hyperactivity disorder and Rett syndrome

Influence of the N-terminus acetylation of Semax, a synthetic analog of ACTH(4-10), on copper(II) and zinc(II) coordination and biological properties

Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents

The efficacy of Semax in the treatment of patients at different stages of ischemic stroke

Effects of Semax on the default mode network of the brain

The heptapeptide SEMAX stimulates BDNF expression in different areas of the rat brain in vivo

Neurotrophin gene expression in rat brain under the action of Semax, an analogue of ACTH 4-10

Comparison of the temporary dynamics of NGF and BDNF gene expression in rat hippocampus, frontal cortex, and retina under Semax action

Semax and Pro-Gly-Pro activate the transcription of neurotrophins and their receptor genes after cerebral ischemia

Brain protein expression profile confirms the protective effect of the ACTH(4-7)PGP peptide (Semax) in a rat model of cerebral ischemia-reperfusion

Novel insights into the protective properties of ACTH(4-7)PGP (Semax) peptide at the transcriptome level following cerebral ischaemia-reperfusion in rats

Semax, an ACTH4-10 peptide analog with high affinity for copper(II) ion and protective ability against metal induced cell toxicity

Semax, a copper chelator peptide, decreases the Cu(II)-catalyzed ROS production and cytotoxicity of Abeta by metal ion stripping and redox silencing

Antidepressant-like and antistress effects of the ACTH(4-10) synthetic analogs Semax and Melanotan II on male rats in a model of chronic unpredictable stress

Semax, synthetic ACTH(4-10) analogue, attenuates behavioural and neurochemical alterations following early-life fluvoxamine exposure in white rats

Semax peptide targets the mu opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female mice

The potential of the peptide drug Semax and its derivative for correcting pathological impairments in the animal model of Alzheimer's disease

Experimental substantiation of application of Semax as a modulator of immune reaction on the model of social stress