Semax

[The study of chronic partial denervation and quality of life in patients with motor neuron disease treated with semax]

[Effectiveness of semax in acute period of hemispheric ischemic stroke (a clinical and electrophysiological study)]

The efficacy of semax in the treatment of patients at different stages of ischemic stroke

Evaluation of therapeutic effect of new Russian drug semax in optic nerve disease

[Neonatal Semax and saline injections induce open-field behavior changes in mice of different genotypes]

Comparative study of modulatory effects of Semax and primary proline-containing peptides on hemostatic reactions

[A nootropic adrenocorticotropin analog 4-10-semax (l5 years experience in its design and study)]

Comparison of anticoagulant effects of regulatory proline-containing oligopeptides

Semax peptide targets the μ opioid receptor gene Oprm1 to promote deubiquitination and functional recovery after spinal cord injury in female mice

The Potential of the Peptide Drug Semax and Its Derivative for Correcting Pathological Impairments in the Animal Model of Alzheimer's Disease

Semax, a Copper Chelator Peptide, Decreases the Cu(II)-Catalyzed ROS Production and Cytotoxicity of aβ by Metal Ion Stripping and Redox Silencing

The Effect of Peptide Semax, an ACTH(4-10) Analogue, on Intracellular Calcium Dynamics in Rat Brain Neurons

Antidepressant-like and antistress effects of the ACTH(4-10) synthetic analogs Semax and Melanotan II on male rats in a model of chronic unpredictable stress

Semax, a Synthetic Regulatory Peptide, Affects Copper-Induced Abeta Aggregation and Amyloid Formation in Artificial Membrane Models

Semax, synthetic ACTH(4-10) analogue, attenuates behavioural and neurochemical alterations following early-life fluvoxamine exposure in white rats

[The Peptide Drug ACTH(4-7)PGP (Semax) Suppresses mRNA Transcripts Encoding Proinflammatory Mediators Induced by Reversible Ischemia of the Rat Brain]

Morphofunctional State of the Large Intestine in Rats under Conditions of Restraint Stress and Administration of Peptide ACTH((4-7))-PGP (Semax)

Brain Protein Expression Profile Confirms the Protective Effect of the ACTH((4-7))PGP Peptide (Semax) in a Rat Model of Cerebral Ischemia-Reperfusion

Functional Connectomic Approach to Studying Selank and Semax Effects

Novel Insights into the Protective Properties of ACTH((4-7))PGP (Semax) Peptide at the Transcriptome Level Following Cerebral Ischaemia-Reperfusion in Rats

Composition of Colon Microbiota in Rats Treated with ACTH(4-7)-PGP Peptide (Semax) under Conditions of Restraint Stress

Peptide ACTH(4-7)-PGP: Effects on Various Types of Pain and Pain-Induced Behavior in Rats after Systemic and Central Administration

Experimental Substantiation of Application of Semax as a Modulator of Immune Reaction on the Model of "Social" Stress

Effects of Semax on the Default Mode Network of the Brain

Modulation of GABA- and Glycine-Activated Ionic Currents with Semax in Isolated Cerebral Neurons

Semax, an analog of ACTH((4-7)), regulates expression of immune response genes during ischemic brain injury in rats

Peptides semax and selank affect the behavior of rats with 6-OHDA induced PD-like parkinsonism

Influence of the N-terminus acetylation of Semax, a synthetic analog of ACTH(4-10), on copper(II) and zinc(II) coordination and biological properties

Semax prevents learning and memory inhibition by heavy metals

Semax, an ACTH4-10 peptide analog with high affinity for copper(II) ion and protective ability against metal induced cell toxicity

Semax, an ACTH(4-10) analogue with nootropic properties, activates dopaminergic and serotoninergic brain systems in rodents